Human papillomavirus-induced carcinogenesis and the ubiquitin–proteasome system

dc.contributor.authorScheffner, Martin
dc.contributor.authorWhitaker, Noel J.
dc.date.accessioned2018-05-16T13:21:18Z
dc.date.available2018-05-16T13:21:18Z
dc.date.issued2003-02eng
dc.description.abstractCertain types of human papillomaviruses have been etiologically associated with malignant lesions, most notably with cervical cancer. The major oncoproteins of these cancer-associated viruses are encoded by the viral E6 and E7 genes. Thorough characterization of these oncoproteins and their interaction with cellular proteins has shown that both E6 and E7 exploit the ubiquitin–proteasome system to degrade and, thus, to functionally inactivate negative cell-regulatory proteins including members of the p110RB family and p53. This act of piracy is assumed to contribute to both the efficient propagation of HPVs and HPV-induced carcinogenesis.eng
dc.description.versionpublishedeng
dc.identifier.doi10.1016/S1044-579X(02)00100-1eng
dc.identifier.urihttps://kops.uni-konstanz.de/handle/123456789/42373
dc.language.isoengeng
dc.subjectCervical cancer; E6-AP; HPV; p53; pRBeng
dc.subject.ddc570eng
dc.titleHuman papillomavirus-induced carcinogenesis and the ubiquitin–proteasome systemeng
dc.typeJOURNAL_ARTICLEeng
dspace.entity.typePublication
kops.citation.bibtex
@article{Scheffner2003-02Human-42373,
  year={2003},
  doi={10.1016/S1044-579X(02)00100-1},
  title={Human papillomavirus-induced carcinogenesis and the ubiquitin–proteasome system},
  number={1},
  volume={13},
  issn={1044-579X},
  journal={Seminars in Cancer Biology},
  pages={59--67},
  author={Scheffner, Martin and Whitaker, Noel J.}
}
kops.citation.iso690SCHEFFNER, Martin, Noel J. WHITAKER, 2003. Human papillomavirus-induced carcinogenesis and the ubiquitin–proteasome system. In: Seminars in Cancer Biology. 2003, 13(1), pp. 59-67. ISSN 1044-579X. eISSN 1096-3650. Available under: doi: 10.1016/S1044-579X(02)00100-1deu
kops.citation.iso690SCHEFFNER, Martin, Noel J. WHITAKER, 2003. Human papillomavirus-induced carcinogenesis and the ubiquitin–proteasome system. In: Seminars in Cancer Biology. 2003, 13(1), pp. 59-67. ISSN 1044-579X. eISSN 1096-3650. Available under: doi: 10.1016/S1044-579X(02)00100-1eng
kops.citation.rdf
<rdf:RDF
    xmlns:dcterms="http://purl.org/dc/terms/"
    xmlns:dc="http://purl.org/dc/elements/1.1/"
    xmlns:rdf="http://www.w3.org/1999/02/22-rdf-syntax-ns#"
    xmlns:bibo="http://purl.org/ontology/bibo/"
    xmlns:dspace="http://digital-repositories.org/ontologies/dspace/0.1.0#"
    xmlns:foaf="http://xmlns.com/foaf/0.1/"
    xmlns:void="http://rdfs.org/ns/void#"
    xmlns:xsd="http://www.w3.org/2001/XMLSchema#" > 
  <rdf:Description rdf:about="https://kops.uni-konstanz.de/server/rdf/resource/123456789/42373">
    <dcterms:available rdf:datatype="http://www.w3.org/2001/XMLSchema#dateTime">2018-05-16T13:21:18Z</dcterms:available>
    <dc:language>eng</dc:language>
    <dcterms:issued>2003-02</dcterms:issued>
    <dc:creator>Whitaker, Noel J.</dc:creator>
    <dspace:isPartOfCollection rdf:resource="https://kops.uni-konstanz.de/server/rdf/resource/123456789/28"/>
    <dc:contributor>Scheffner, Martin</dc:contributor>
    <dc:contributor>Whitaker, Noel J.</dc:contributor>
    <dcterms:isPartOf rdf:resource="https://kops.uni-konstanz.de/server/rdf/resource/123456789/28"/>
    <dcterms:abstract xml:lang="eng">Certain types of human papillomaviruses have been etiologically associated with malignant lesions, most notably with cervical cancer. The major oncoproteins of these cancer-associated viruses are encoded by the viral E6 and E7 genes. Thorough characterization of these oncoproteins and their interaction with cellular proteins has shown that both E6 and E7 exploit the ubiquitin–proteasome system to degrade and, thus, to functionally inactivate negative cell-regulatory proteins including members of the p110RB family and p53. This act of piracy is assumed to contribute to both the efficient propagation of HPVs and HPV-induced carcinogenesis.</dcterms:abstract>
    <dc:creator>Scheffner, Martin</dc:creator>
    <void:sparqlEndpoint rdf:resource="http://localhost/fuseki/dspace/sparql"/>
    <bibo:uri rdf:resource="https://kops.uni-konstanz.de/handle/123456789/42373"/>
    <dc:date rdf:datatype="http://www.w3.org/2001/XMLSchema#dateTime">2018-05-16T13:21:18Z</dc:date>
    <dcterms:title>Human papillomavirus-induced carcinogenesis and the ubiquitin–proteasome system</dcterms:title>
    <foaf:homepage rdf:resource="http://localhost:8080/"/>
  </rdf:Description>
</rdf:RDF>
kops.flag.knbibliographyfalse
kops.sourcefieldSeminars in Cancer Biology. 2003, <b>13</b>(1), pp. 59-67. ISSN 1044-579X. eISSN 1096-3650. Available under: doi: 10.1016/S1044-579X(02)00100-1deu
kops.sourcefield.plainSeminars in Cancer Biology. 2003, 13(1), pp. 59-67. ISSN 1044-579X. eISSN 1096-3650. Available under: doi: 10.1016/S1044-579X(02)00100-1deu
kops.sourcefield.plainSeminars in Cancer Biology. 2003, 13(1), pp. 59-67. ISSN 1044-579X. eISSN 1096-3650. Available under: doi: 10.1016/S1044-579X(02)00100-1eng
relation.isAuthorOfPublication5c3398d2-7e1e-413c-9edf-b32e9d9fdf82
relation.isAuthorOfPublication.latestForDiscovery5c3398d2-7e1e-413c-9edf-b32e9d9fdf82
source.bibliographicInfo.fromPage59eng
source.bibliographicInfo.issue1eng
source.bibliographicInfo.toPage67eng
source.bibliographicInfo.volume13eng
source.identifier.eissn1096-3650eng
source.identifier.issn1044-579Xeng
source.periodicalTitleSeminars in Cancer Biologyeng

Dateien