Publikation:

Poly(ADP-ribosyl)ation in mammalian ageing

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2007_NucleicAcidsRes_Beneke.pdf
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2007

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http://nbn-resolving.de/urn:nbn:de:bsz:352-opus-76253
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https://doi.org/10.1126/sageke.2004.49.re9
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Nucleic Acids Research. 2007, 35(22), pp. 7456-7465. eISSN 1539-6150. Available under: doi: 10.1126/sageke.2004.49.re9

Zusammenfassung

Poly(ADP-ribose) polymerases (PARPs) catalyze the post-translational modification of proteins with poly(ADP-ribose). Two PARP isoforms, PARP-1 and PARP-2, display catalytic activity by contact with DNA-strand breaks and are involved in DNA baseexcision repair and other repair pathways. A body of correlative data suggests a link between damage-induced poly(ADP-ribosyl)ation and mammalian longevity. Recent research on PARPs and poly(ADP-ribose) yielded several candidate mechanisms through which poly(ADP-ribosyl)ation might act as a factor that limits the rate of ageing.

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570 Biowissenschaften, Biologie

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ISO 690BENEKE, Sascha, Alexander BÜRKLE, 2007. Poly(ADP-ribosyl)ation in mammalian ageing. In: Nucleic Acids Research. 2007, 35(22), pp. 7456-7465. eISSN 1539-6150. Available under: doi: 10.1126/sageke.2004.49.re9
BibTex
@article{Beneke2007PolyA-6615,
  year={2007},
  doi={10.1126/sageke.2004.49.re9},
  title={Poly(ADP-ribosyl)ation in mammalian ageing},
  number={22},
  volume={35},
  journal={Nucleic Acids Research},
  pages={7456--7465},
  author={Beneke, Sascha and Bürkle, Alexander}
}
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