Publikation: Activation of zebrafish Src family kinases by the prion protein is an amyloid-β-sensitive signal that prevents the endocytosis and degradation of E-cadherin/β-catenin complexes in vivo
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2016
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Molecular Neurodegeneration. 2016, 11, 18. eISSN 1750-1326. Available under: doi: 10.1186/s13024-016-0076-5
Zusammenfassung
Prions and amyloid-β (Aβ) oligomers trigger neurodegeneration by hijacking a poorly understood cellular signal mediated by the prion protein (PrP) at the plasma membrane. In early zebrafish embryos, PrP-1-dependent signals control cell-cell adhesion via a tyrosine phosphorylation-dependent mechanism.
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570 Biowissenschaften, Biologie
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SEMPOU, Aimilia, Emiliano BIASINI, Alejandro PINZÓN-OLEJUA, David A. HARRIS, Edward MÁLAGA-TRILLO, 2016. Activation of zebrafish Src family kinases by the prion protein is an amyloid-β-sensitive signal that prevents the endocytosis and degradation of E-cadherin/β-catenin complexes in vivo. In: Molecular Neurodegeneration. 2016, 11, 18. eISSN 1750-1326. Available under: doi: 10.1186/s13024-016-0076-5BibTex
@article{Sempou2016-12Activ-33640,
year={2016},
doi={10.1186/s13024-016-0076-5},
title={Activation of zebrafish Src family kinases by the prion protein is an amyloid-β-sensitive signal that prevents the endocytosis and degradation of E-cadherin/β-catenin complexes in vivo},
volume={11},
journal={Molecular Neurodegeneration},
author={Sempou, Aimilia and Biasini, Emiliano and Pinzón-Olejua, Alejandro and Harris, David A. and Málaga-Trillo, Edward},
note={Article Number: 18}
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<dc:contributor>Sempou, Aimilia</dc:contributor>
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<dc:creator>Biasini, Emiliano</dc:creator>
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